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Blood glucose response after oral lactulose intake in type 2 diabetic individuals. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4. Published by Baishideng Publishing Group Inc. Institutional review board statement: This study protocol was Omeclamox-Pak (Omeprazole Delayed-release Capsules)- Multum and approved by the Independent Ethics Committee of the Medical University of Graz, Austria. Clinical trial registration statement: This study was registered in the European Union Drug Regulating Authorities Clinical Trials Database, No.

Informed consent statement: All study participants provided written informed consent prior to enrollment. Conflict-of-interest statement: The Omeclamox-Pak (Omeprazole Delayed-release Capsules)- Multum was sponsored by Fresenius Kabi Deutschland GmbH, Germany. All other authors declare no potential conflicts of interest related to this paper. CONSORT 2010 statement: The authors read the CONSORT 2010 Statement, Omeclamox-Pak (Omeprazole Delayed-release Capsules)- Multum the manuscript was prepared and revised according to that statement.

Open-Access: This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4. Citation: Pieber TR, Svehlikova E, Mursic I, Esterl T, Wargenau M, Sartorius T, Pauly L, Schwejda-Guettes S, Neumann A, Faerber V, Stover JF, Gaigg B, Kuchinka-Koch A. Lactulose vs negative control (water) Figure 2 Blood glucose concentration-time curves.

Table 2 Blood glucose endpoints: 20 g crystal lactulose vs water. Table 3 Blood glucose endpoints: 30 g crystal lactulose vs Omeclamox-Pak (Omeprazole Delayed-release Capsules)- Multum. Table 4 Blood glucose endpoints: 20 g liquid lactulose vs water. Figure 3 Cumulative numbers of bowel movements per treatment and time after oral intake of water, glucose, or lactulose for all subjects. Manuscript source: Unsolicited manuscriptCorresponding Author's Membership in Professional Societies: American Society for Parenteral and Enteral Nutrition (ASPEN), No.

Zhao M, Liao D, Zhao J. Diabetes-induced mechanophysiological changes in the small intestine and colon. Bytzer P, Talley NJ, Hammer J, Young LJ, Jones MP, Horowitz M.

GI symptoms in diabetes mellitus are associated with both poor glycemic control and Albumin (Human) USP, 5% Solution (Flexbumin)- FDA complications. The possible role of the gut neuroendocrine system in diabetes gastroenteropathy. Rayner CK, Samsom M, Jones KL, Horowitz M. Ricci JA, Siddique R, Stewart WF, Sandler RS, Sloan S, Farup CE. Upper gastrointestinal symptoms in a U. Chandrasekharan B, Anitha M, Blatt R, Shahnavaz N, Kooby D, Staley C, Mwangi S, Jones DP, Sitaraman SV, Srinivasan S.

Piper MS, Saad RJ. Diabetes Mellitus and the Colon. Curr Treat Options Gastroenterol. Prasad VG, Abraham P. Management of chronic constipation in patients with diabetes mellitus. Cash BD, Lacy BE. Systematic Review: FDA-Approved Prescription Medications for Adults With Constipation.

Gastroenterol Hepatol (N Y). Portalatin M, Winstead N. Medical management of constipation. Clin Colon Rectal Surg. Shakil A, Church RJ, Rao SS. Gastrointestinal complications of diabetes. Current treatment options for chronic constipation.

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